Basal cell carcinoma
Signs and symptoms
Basal cell carcinoma is usually a disease of white people, especially those with thin skin. It is rare in black people. Ratio men: women is 3:2. Appears more common in adults especially in older people. Patients often have an ulcer that will not heal the duration variable. Typical lesion is seen on the face, ears, scalp, neck or upper trunk. Trauma easy as washing your face and towel dry initially, can cause bleeding. It often describes a personal history of occupational or recreational sun exposure. Intense sun exposure occurs in childhood or adolescence.
Physical examination. Several clinical subtypes may hidtologice different behaviors. Recognize the different types is important because it is often necessary for aggressive terapis ariante such as micronodular, infiltrative basal cell carcinoma or morfeoforma. When you are examining a possible skin cancer is advisable to use good lighting and optical enhancement. Affected skin must be stretched to estimate palpable tumor size and depth. Oblique illumination of the tumor may exhibit surface changes as supradenivelata edge.
Spheroidal cell carcinoma. It is the most common variants of basal cell carcinoma. Appears frequently on the head, neck and back. Features: -Waxy papules with central depression, pearly appearance Erosion or ulceration, bleeding, crustificare Supradenivelata-edge, translucent, surface teleangiectazii -History of bleeding from minor trauma.
Basal cell carcinoma Signs and symptoms Pigmented basal cell carcinoma. Unlike the variety characteristics of this variety nodular lesions contain brown or black pigment and are common in people with darker skin.
Basal cell carcinoma Signs and symptoms Cystic basal cell carcinoma. Cystic basal cell carcinoma lesions are translucent blue-gray cystic nodules that may mimic benign cysts.
Superficial basal cell carcinoma. This variety appears as scaly patches or papules red-red-brown, often with central fading. Erosion is less common in this form from the nodular. Superficial basal cell carcinoma is frequently on the trunk and has a low tendency to become invasive. Papules may mimic psoriasis or eczema, but are slowly progressive and does not predispose to an aspect fluctuent. Many tumors may indicate expuenrea to arsenic.
Micronodular basal cell carcinoma. It is an aggressive form of basal cell carcinoma of the typical distribution. It is prone to ulceration, may be yellow-white when it is stretched and is firm to touch. Maybe this a well-defined edges.
Basal cell carcinoma and infiltrating morfeaform. Sclerotic plates are aggressive subtypes and papules. The edge is usually defined and bad seextinde clinic over the edge. Ulceration, bleeding and crustificarea are rare. Can be confused with a scar.
Monday, January 31, 2011
Bowen disease Treatment
Bowen disease
Treatment
Medical therapy. Topical administration of 5-fluorouracil under occlusion, followed by a keratolytic or cryosurgery is an effective regime in Bowen's disease. 5% imiquimod cream, a topical immune response modifier seems to be a successful treatment for Bowen's disease with multiple locations. Photodynamic therapy shows promising results in treating superficial carcinomas. It involves introducing a photosensitizing agent in the body, preferentially retained by the tumor. Then the affected area is exposed to blue light to the active agent, causes the release of toxins and destroy the tumor.
Surgical therapy. Simple excision with conventional margins. It is the technique used for small lesions that are not problematic areas: the face and fingers. Although the lesions are well demarcated, the current expansion of clinical disease can be ejected, so excision must provide safety margins of 4 mm.
Bowen disease Treatment Mohs micrographic surgery. It is an excellent method for large lesions, recurring or from areas where the tissue is important. Use systematic surgical removal of cancer tissue with small mergini safety and frozen sectioned for examination. Safety margins are 100% negative for carcinoma.
Electrodesicarea and curettage, cryotherapy and laser ablation of carbon. These surgical methods are blind, without pathological confirmation of the removal of carcinoma.
Bowen disease Treatment Prognosis. The prognosis is excellent for all forms of disease. Cure rate is 90%
Treatment
Medical therapy. Topical administration of 5-fluorouracil under occlusion, followed by a keratolytic or cryosurgery is an effective regime in Bowen's disease. 5% imiquimod cream, a topical immune response modifier seems to be a successful treatment for Bowen's disease with multiple locations. Photodynamic therapy shows promising results in treating superficial carcinomas. It involves introducing a photosensitizing agent in the body, preferentially retained by the tumor. Then the affected area is exposed to blue light to the active agent, causes the release of toxins and destroy the tumor.
Surgical therapy. Simple excision with conventional margins. It is the technique used for small lesions that are not problematic areas: the face and fingers. Although the lesions are well demarcated, the current expansion of clinical disease can be ejected, so excision must provide safety margins of 4 mm.
Bowen disease Treatment Mohs micrographic surgery. It is an excellent method for large lesions, recurring or from areas where the tissue is important. Use systematic surgical removal of cancer tissue with small mergini safety and frozen sectioned for examination. Safety margins are 100% negative for carcinoma.
Electrodesicarea and curettage, cryotherapy and laser ablation of carbon. These surgical methods are blind, without pathological confirmation of the removal of carcinoma.
Bowen disease Treatment Prognosis. The prognosis is excellent for all forms of disease. Cure rate is 90%
Bowen disease Symptoms and Diagnosis
Bowen disease
Symptoms and Diagnosis
Bowen's disease has a high incidence in the elderly. Asymptomatic patients have a spot, slowly progressive erythematous with scaling. It can occur anywhere on the mucocutaneous surface. It shows that a single lesion or multiple. It can occur on sun-exposed areas covered. Head and neck are the most common areas affected, followed by States. Lesions vary in size from several mm to cm. is this a well-demarcated border, irregular. The lesions are erythematous, with scales, or plates may become hyperkeratotic, scaly, cracking or ulceration. Pigmented lesions are rare, especially in the genital area and nails. These lesions can simulate melanoma. Bowen's disease can occur on mucous areas.
Penis form of the disease. Mean age of onset is 51 years. Characteristic lesions are solitary or multiple erythematous boards. Texture can be smooth, velvety, scaly or warty. Always affects the glans penis or adjacent mucosal surfaces, or both. The lesions may represent forms of carcinoma in situ induced by papilloma virus. Representative Symptoms include: -Erythema, crustificare, desquamation, ulceration -Bleeding, pain, itching, dysuria -Debacluri penis, difficulty in decalotare.
Bowen disease Symptoms and Diagnosis
Laboratory studies: By tangential shaving skin-biopsy or fine needle puncture to confirm the diagnosis -Culture bacterial, fungal, Tzanck test, examination with potassium hydroxide, Gram stain, the shape penis.
Bowen disease Symptoms and Diagnosis Histological examination. Bowen's disease is an anaplastic the entire thickness of the epidermis, with loss of normal maturation of its components. Keratinocytes are atypical and in disarray, described as blown by the wind. Basal layer is intact. Vacuolation are present in epidermis, mitoses, individual keratinized cells, multinucleated cells. Superficial dermis shows a moderate degree of infiltrating lymphoma. The differential diagnosis is made with the following conditions: Balan xerotica Peripheral, balanopostita, mucosal candidiasis, contact dermatitis, bullous diseases induced by drugs, boards psoriasis, actinic keratosis, basal cell carcinoma, lichen simplex cronicus, Paget's disease, tinea corporis.
Symptoms and Diagnosis
Bowen's disease has a high incidence in the elderly. Asymptomatic patients have a spot, slowly progressive erythematous with scaling. It can occur anywhere on the mucocutaneous surface. It shows that a single lesion or multiple. It can occur on sun-exposed areas covered. Head and neck are the most common areas affected, followed by States. Lesions vary in size from several mm to cm. is this a well-demarcated border, irregular. The lesions are erythematous, with scales, or plates may become hyperkeratotic, scaly, cracking or ulceration. Pigmented lesions are rare, especially in the genital area and nails. These lesions can simulate melanoma. Bowen's disease can occur on mucous areas.
Penis form of the disease. Mean age of onset is 51 years. Characteristic lesions are solitary or multiple erythematous boards. Texture can be smooth, velvety, scaly or warty. Always affects the glans penis or adjacent mucosal surfaces, or both. The lesions may represent forms of carcinoma in situ induced by papilloma virus. Representative Symptoms include: -Erythema, crustificare, desquamation, ulceration -Bleeding, pain, itching, dysuria -Debacluri penis, difficulty in decalotare.
Bowen disease Symptoms and Diagnosis
Laboratory studies: By tangential shaving skin-biopsy or fine needle puncture to confirm the diagnosis -Culture bacterial, fungal, Tzanck test, examination with potassium hydroxide, Gram stain, the shape penis.
Bowen disease Symptoms and Diagnosis Histological examination. Bowen's disease is an anaplastic the entire thickness of the epidermis, with loss of normal maturation of its components. Keratinocytes are atypical and in disarray, described as blown by the wind. Basal layer is intact. Vacuolation are present in epidermis, mitoses, individual keratinized cells, multinucleated cells. Superficial dermis shows a moderate degree of infiltrating lymphoma. The differential diagnosis is made with the following conditions: Balan xerotica Peripheral, balanopostita, mucosal candidiasis, contact dermatitis, bullous diseases induced by drugs, boards psoriasis, actinic keratosis, basal cell carcinoma, lichen simplex cronicus, Paget's disease, tinea corporis.
Preventing sunburn
Preventing sunburn
Prevention is the most effective therapy for sunburn. Effective individual and community educational programs can reduce sun exposure and increased use of creams or protective clothing. Avoid sun exposure during the maximum 10-hour solar radiation. oo-16. oo day.
It indicates the use of sunscreens with SPF-creams suitable for your skin type. SPF is the amount of ultraviolet energy required to produce erythema on protected skin to the amount of ultraviolet energy required to produce erythema on unprotected skin. Apply for at least 30 minutes before sun exposure and reapply every 2-3 hours or after swimming, sweating or shower. Apply before exposure of small children. It will use water-resistant SPF creams. will apply the cream at least 2 mg/cm2 to reach the SPF. Most people apply a concime of that amount.
Physical barriers (zinc oxide, titanium diozid) allow excellent protection against UV-A and UV-B and are photostat. Chemical barriers are used in most solar screenings. Paraaminobenzenic acid and esters are not indicated due to high rates of contact dermatitis associated. Other UVB blockers include salicylates and cinamates. Avobenzona contain UVA blockers, and terefitalidena drometizole trisiloxane.
Preventing sunburn
Wearing protective clothing, including hats and sunglasses ocgelari. Clothing can be treated to increase the product's SPF. Dark clothes, dried offers the highest protection. When the fiber is wet, water occupies the space between fibers and allows entry ultraviolet. Nylon clothing offers the lowest protection. Synthetic fabrics, Raiatea offers more protection from cotton. People are encouraged to use self-tanning lotions and creams that do not increase the risk of skin cancer or wrinkles. They offer sun protection. Avoid tanning salons. Avoid phototoxic administration of drugs or toxic agents that can interact with sunlight to avoid burning.
Pharmacological aspects of prevention include screening chemical and solar physics. No sunscreen does not confer 100% protection. There is no known effective oral sunscreen. Antimalarial drugs, vitamins A, E, beta carotene and oral paraaminobenzenic acid does not provide adequate sun protection. Systemic administration of vitamin C and E combined-SPF provides a minimum protection. 4. Solar screens are designed to protect the skin and not to prolong sun exposure. Although they can prevent burns, and immunosuppression caused by ultraviolet carcinogenicity is not avoided. It is better to use creams that block SPF 15 or 93% of UVB rays with SPF 30 to 97% blocking.
Prognosis sunburn
Preventing sunburn Sunburn uncomplicated resolve spontaneously within 4-7 days without sequelae acute desquamation. Sunburn can exacerbate chronic diseases: chronic actinic dermatitis, herpes simplex, lupus erythematosus, eczema. May be associated with other diseases caused by heat, including stroke and dehydration. Long term exposure leads to premature formation of wrinkles, fotoimbatrinire, development of premalignant lesions, solar keratoses, development of malignancies: basal cell carcinoma, squamous cell carcinoma and malignant melanoma.
Sunburn treatment
Sunburn treatment
A sunburn is considered a self-limiting lesion. But even a slight burn that does not require treatment for these patients is an opportunity to use and explore the consequences of UV photoprotection. Patients with moderate burns should be encouraged to hydrate with non-alcoholic abuturi. Those with moderate to severe burning pain and itch requires. Required in severe burns ward intenare reinlocuirea burned for parenteral fluids, pain control and prevention of infections. Patients with vesicular recatie should not break the skin surface of the gall bladder because it provides protection against infection. After a skin sunburn should not be exposed to sunlight for at least a week because the skin is succeptibila to burn.
Sunburn treatment
Nonarmacologic treatment for sunburn include: Intruhn-average patient stay cool, covered for several days, with rest in bed if necessary -Cold baths or applying cold towels or compression with saline for 20 minutes and repeated 3-4 times per day may provide relief of pain and itch Promoxina soot-application or 3 times a day on affected areas can help Creams applied repeatedly, helps reduce dryness and desquamation in the light burns -Oil or butter does not help moderate burns can be painful and washed.
Medical therapy. Topical vitamin E applied to two minutes to excessive sun exposure can reduce the redness and swelling. However, because the clinical effects of burns do not occur until after a certain time of exposure, the risk of sunburn may not be appreciated at the time of exposure. Oral indomethacin or ibuprofen or topical 1% indomethacin administered immediately after exposure to sunlight may reduce the degree of erythema and epidermal injury. Diclofenac gel applied 6 hours and 10 hours after irradiation can relieve pain and decreases spontaneous and induced erythema, edema and skin temperature. The effect lasts 48 hours after application.
Application of 12% acid glycine decreases erythema topic. Three days of pretreatment with acid glycine determine an SPF of 24. Systemic corticosteroids have little benefit in treating burns and may increase the risk of secondary infection. Potent topical corticosteroids reduce transient erythema but does not influence skin damage. Tacrolimus topic has no significant effect on erythema induced by sunlight or inflammation administered before exposure.
Sunburn treatment
Acetaminophen or aspirin may provide a degree of pain relief, although opioid analgesics may be necessary in severe sitiatii. Diphenhydramine or hydroxyzine may help patients fall asleep and improves priuritul. Diphenhydramine did not indicate the use of topical or topical anesthetic spray because of the risk of allergic contact dermatitis.
Sunday, January 30, 2011
Sunburn Diagnosis
Sunburn Diagnosis
Laboratory studies: Burn-in if they are not necessary If porphyria is suspected, obtain blood tests, urine and stool porphyrins in -If it is suspected systemic lupus erythematosus antinuclear antibody test are obtained, anti-Ro and anti-La -Photosensitivity can be tested by skin test to determine an allergen fotocontact Fototestarea may show erythema-response decrease actinic dermatitis -If secondary infection is suspected microbiological tests are obtained.
Sunburn Diagnosis
Histological examination helps to distinguish between sunburn and forosensibilitate. In patients with skin examination showed sunburn burns and vacuole diskeratinice keratinocyte cells with pyknotic nuclei, sponginess nminima, vacuolation of melanocytes and decreased number of Langerhans cells. Examination shows dermal mast cell degranulation, endothelial cells of the plexus balonizarea superficial and deep skin and a mixed perivascular infiltrate. Endothelial Balonizarea is apparent within 30 minutes after irradiation with a peak at 24 hours.
Sunburn Diagnosis
The differential diagnosis is made with the following conditions: Arsu chemical, electrical burns, thermal burns, toxic shock syndrome, drug photosensitivity, dermatomyositis, drug-induced lupus erythematosus, polymorphic light eruption, protoporfiria erythropoietic, chronic actinic dermatitis.
Laboratory studies: Burn-in if they are not necessary If porphyria is suspected, obtain blood tests, urine and stool porphyrins in -If it is suspected systemic lupus erythematosus antinuclear antibody test are obtained, anti-Ro and anti-La -Photosensitivity can be tested by skin test to determine an allergen fotocontact Fototestarea may show erythema-response decrease actinic dermatitis -If secondary infection is suspected microbiological tests are obtained.
Sunburn Diagnosis
Histological examination helps to distinguish between sunburn and forosensibilitate. In patients with skin examination showed sunburn burns and vacuole diskeratinice keratinocyte cells with pyknotic nuclei, sponginess nminima, vacuolation of melanocytes and decreased number of Langerhans cells. Examination shows dermal mast cell degranulation, endothelial cells of the plexus balonizarea superficial and deep skin and a mixed perivascular infiltrate. Endothelial Balonizarea is apparent within 30 minutes after irradiation with a peak at 24 hours.
Sunburn Diagnosis
The differential diagnosis is made with the following conditions: Arsu chemical, electrical burns, thermal burns, toxic shock syndrome, drug photosensitivity, dermatomyositis, drug-induced lupus erythematosus, polymorphic light eruption, protoporfiria erythropoietic, chronic actinic dermatitis.
Sunburn Signs and symptoms
Sunburn
Signs and symptoms
Men frequently experience sunburn, using less protection and tend to be less knowledgeable than women about the effects of ultraviolet light. Although solareapar burns to people of all ages, the incidence increases in childhood and adolescence. The highest prevalence is recorded between 15-24 years.
Patients with a history of sunburn shows excessive sun exposure by acitivitati in free space and less history of concomitant oral ingestion of drugs or application of topical phototoxic. All individuals develop high doses of ultraviolet burns although people with darker skin are more protected. Perosanele blue or green eyes, white skin who tan poorly are more likely to burn.
If the patient received late erythema dose of UV exposed skin occurs in 2-6 hours with a peak at 15-36 hours and resolve within 72-120 hours. People with thin skin may be transient erythema immediately. Trunk, neck and face injuries at doses lower than upper limbs. Upper limbs tan faster towards lower ones. Erythema after exposure to solar UV-A by starting immediately, with a peak at 8 hours and persists for 24-48 hours. Patients with fever, local pain, veziculizare, erythema that resolves in 4-7 days with burned skin peeling, malaise, nausea and vomiting in severe cases.
The physical examination includes: Confluent erythema and heat-exposed areas of the local -Edema, pain and tenderness, with moderate to severe itching at the exposure -Veziculizarea occurs in severe cases and can require over a week to submit -Scaling occurs in a few days after exposure -Nausea, abdominal cramps, weakness and malaise, fever, chills and headache may occur, most commonly in severe burns By secondary infection, complications are infrequent.
Sunburn
Signs and symptoms
Complications. Sunburn can exacerbate diseases such as chronic actinic dermatitis, herpes simplex, eczema, and lupus erythematosus. Sunburn can be associated with other diseases related to heat, dehydration and heat stroke exhaustierea through. Long term exposure can lead to premature aging skin and wrinkles-dermatohelioza training, development of solar keratoses, premalignant lesions and malignant tumors developing basal-cell carcinoma, squamous cell carcinoma and malignant melanoma. Patients may be at risk by ultraviolet keratitis and cataracts.
Signs and symptoms
Men frequently experience sunburn, using less protection and tend to be less knowledgeable than women about the effects of ultraviolet light. Although solareapar burns to people of all ages, the incidence increases in childhood and adolescence. The highest prevalence is recorded between 15-24 years.
Patients with a history of sunburn shows excessive sun exposure by acitivitati in free space and less history of concomitant oral ingestion of drugs or application of topical phototoxic. All individuals develop high doses of ultraviolet burns although people with darker skin are more protected. Perosanele blue or green eyes, white skin who tan poorly are more likely to burn.
If the patient received late erythema dose of UV exposed skin occurs in 2-6 hours with a peak at 15-36 hours and resolve within 72-120 hours. People with thin skin may be transient erythema immediately. Trunk, neck and face injuries at doses lower than upper limbs. Upper limbs tan faster towards lower ones. Erythema after exposure to solar UV-A by starting immediately, with a peak at 8 hours and persists for 24-48 hours. Patients with fever, local pain, veziculizare, erythema that resolves in 4-7 days with burned skin peeling, malaise, nausea and vomiting in severe cases.
The physical examination includes: Confluent erythema and heat-exposed areas of the local -Edema, pain and tenderness, with moderate to severe itching at the exposure -Veziculizarea occurs in severe cases and can require over a week to submit -Scaling occurs in a few days after exposure -Nausea, abdominal cramps, weakness and malaise, fever, chills and headache may occur, most commonly in severe burns By secondary infection, complications are infrequent.
Sunburn
Signs and symptoms
Complications. Sunburn can exacerbate diseases such as chronic actinic dermatitis, herpes simplex, eczema, and lupus erythematosus. Sunburn can be associated with other diseases related to heat, dehydration and heat stroke exhaustierea through. Long term exposure can lead to premature aging skin and wrinkles-dermatohelioza training, development of solar keratoses, premalignant lesions and malignant tumors developing basal-cell carcinoma, squamous cell carcinoma and malignant melanoma. Patients may be at risk by ultraviolet keratitis and cataracts.
Sunburn Causes and Risk Factors
Sunburn
Causes and Risk Factors
Factors affecting UV-induced erythema include: Wave-length, UV-B is more than UV-A eritmogen -Pigmentation and skin type, people with a more pronounced pigmentation, Brunetière and blacks are more protected from burns, compared to whites, and people with oily or combination skin type to those with dry skin -Hydration-ultraviolet burns more frequently cause skin moist from the dry -Reflection-radiation environment is reflected by 80% compared with ice and snow, sand 15% High-ozone layer more UV filters -Altitude, thin air and is present at high altitudes-up to absorb less-beam exposure is greater at the equator The day-time UV exposure is high-between 10 am and 4 day, when the sun is at its zenith (in the middle of the sky).
Phototoxic reactions are caused by topical and systemic pharmacologic agents, cause sunburn UV-A spectrum. In contrast with photoallergic reactions, phototoxic reactions may occur after initial exposure of the body and does not affect areas that were protected from light.
Topical agents that cause phototoxicity include: Coal-derivatives-acridine, anthracene, fluorentenul, naphthalene, phenanthrene, pyridine, thiophene, - Pigments, paint-acriflavina, antraquinona, cadmium sulfate, eosin, methylene blue, rose Bengal, blue toluidin -Metoxsalen Legiminoaselor-plant-family, family Moraceae, Rutaceae family-poratocala, lemon, lime Umbeliferae-family-, retinoids, tazarotene, adapalene, vitamin A.
Systemic agents that cause phototoxicity include: -Antimicrobial: ceftazidime, griseofulvin, quinolones, sulfonamides, tetracyclines, trimetroprimul -Antineoplazicele: dacarbazine, 5-fluorouracil, vinblastine -Antimalarials: quinina Cadiace-drugs: quinidine, amiodarone -Diuretics: furosemide, hydrochlorothiazide -Lipid: clifobrat, atorvastatin Nonsteroidal anti-inflammatory, diclofenac, ibuprofen, indomethacin, ketoprofen, naproxen, piroxicam, sulindac -Psoralen: metoxsalen, triosalen -Antipsychotics: alprazolam, chlorpromazine, despiramina, imipramine, perfanazina, tioridazone -Retinoids: acitretin, isotretinoin -Sulfonyl-urea tolbutamide.
Sunburn
Causes and Risk Factors
Causes and Risk Factors
Factors affecting UV-induced erythema include: Wave-length, UV-B is more than UV-A eritmogen -Pigmentation and skin type, people with a more pronounced pigmentation, Brunetière and blacks are more protected from burns, compared to whites, and people with oily or combination skin type to those with dry skin -Hydration-ultraviolet burns more frequently cause skin moist from the dry -Reflection-radiation environment is reflected by 80% compared with ice and snow, sand 15% High-ozone layer more UV filters -Altitude, thin air and is present at high altitudes-up to absorb less-beam exposure is greater at the equator The day-time UV exposure is high-between 10 am and 4 day, when the sun is at its zenith (in the middle of the sky).
Phototoxic reactions are caused by topical and systemic pharmacologic agents, cause sunburn UV-A spectrum. In contrast with photoallergic reactions, phototoxic reactions may occur after initial exposure of the body and does not affect areas that were protected from light.
Topical agents that cause phototoxicity include: Coal-derivatives-acridine, anthracene, fluorentenul, naphthalene, phenanthrene, pyridine, thiophene, - Pigments, paint-acriflavina, antraquinona, cadmium sulfate, eosin, methylene blue, rose Bengal, blue toluidin -Metoxsalen Legiminoaselor-plant-family, family Moraceae, Rutaceae family-poratocala, lemon, lime Umbeliferae-family-, retinoids, tazarotene, adapalene, vitamin A.
Systemic agents that cause phototoxicity include: -Antimicrobial: ceftazidime, griseofulvin, quinolones, sulfonamides, tetracyclines, trimetroprimul -Antineoplazicele: dacarbazine, 5-fluorouracil, vinblastine -Antimalarials: quinina Cadiace-drugs: quinidine, amiodarone -Diuretics: furosemide, hydrochlorothiazide -Lipid: clifobrat, atorvastatin Nonsteroidal anti-inflammatory, diclofenac, ibuprofen, indomethacin, ketoprofen, naproxen, piroxicam, sulindac -Psoralen: metoxsalen, triosalen -Antipsychotics: alprazolam, chlorpromazine, despiramina, imipramine, perfanazina, tioridazone -Retinoids: acitretin, isotretinoin -Sulfonyl-urea tolbutamide.
Sunburn
Causes and Risk Factors
Acne Important!
Acne
Acne is a disease with a wide variety clinic, whose seriousness is sometimes difficult to diagnose even by some dermatologists.
Acne is an important psychological involvement.
The patient must be addressed immediately dermatologist at the smallest signs of disease.
In no event will address these patients laboratories "autovaccinuri factory. Autovaccinurile not treat acne but may worsen the immune system and change !!!!!
In no event will address patients' cosmeticienelor "
Aneea be treated by dermatologists sometimes team endocrinologist, gynecologist, painter and psychiatrist.
Dermatologist must have experience handling and ease of retinoids and generally use them in the first intention is to use either their local or per os.
Acne is a disease with a wide variety clinic, whose seriousness is sometimes difficult to diagnose even by some dermatologists.
Acne is an important psychological involvement.
The patient must be addressed immediately dermatologist at the smallest signs of disease.
In no event will address these patients laboratories "autovaccinuri factory. Autovaccinurile not treat acne but may worsen the immune system and change !!!!!
In no event will address patients' cosmeticienelor "
Aneea be treated by dermatologists sometimes team endocrinologist, gynecologist, painter and psychiatrist.
Dermatologist must have experience handling and ease of retinoids and generally use them in the first intention is to use either their local or per os.
Acne Particular cases of acne treatment
Acne
Particular cases of acne treatment
Acne fulminans Although this rare form of ancnee may be encountered in practice in general dermatology with a high frequency of males between 13 and 18. Onset is abrupt at presternal, back and shoulders, with hemorrhagic ulcerative lesions, necrotic, fever, pol, and altered mental status ialtralgii General Biology finds proteinuria, circulating immune complexes, polinucleoza.Uneori progression to acne fulminans may be triggered by treatment isotretinoin, in which case corticosteroid therapy should be started at a dose necessary intrerupt.Este 0.5-1mg per kg body weight per zi.Unii author's association with NSAIDs or paracetamolului.Reintroducerea isotretinoin is gradual, with doses of 5-10 mg / kg of body is not possible reintroduction of isotretinoin zi.Daca can call on spironolactona.Foarte disulona or local measures are important in the vacuum treatment of cystic necrotic lesions, and especially psychological support. (2,3,6,)
Foliculitele with Gram negative They are the only cases that require sampling for culture and antibiogram according to which antibiotic to prescribe.
Piodermitele facial Occur especially in women with inflammatory abrupt onset, especially in the nose and obrajilor.Poate require general corticotherapy (0.5-1mg / kg), followed by NSAIDs, which are preferred by isotretinoin with higher anti-inflammatory effect ciclinelor.
Acne Particular cases of acne treatment Acne Conglobata Acne Conglobata represent 0.5-1% of cases of acne and is especially important for men with seborrhea. Because this type of acne cysts and tunnels which prevails out pus, and scarring are important, they must first be intent isotretinoin therapy (0.5-1mg / kg). Sometimes associated with corticosteroid therapy (0.5 mg / kg) and treatment of acne dermatochirurgical.Tratamentul in this form with your team to do plastician.Pacientul may need psychological support! (2,3,6)
Acne Escorial More common in women with psychiatric disorders or other neurotic tendency, this type of acne is well treated in collaboration with the psychiatrist. (Anxiolytics can be prescribed provided that the patient to accept collaboration with psychiatrist or psychologist).
Atrophic or hypertrophic scars of acne may benefit from peels to improve the appearance and performance chemicals pielii.Alegerea peels are done only by experienced dermatologists and plasticienii. (2,3,8)
Acne Particular cases of acne treatment Resistant or relapsing acne to isotretinoin It is generally caused by: 1) hereditary land hiperseboreic with parents acne (m2 mutations demonstrated the existence of an adenine-guanine instead of the cytochrome P450 gene in subjects acne, which leads to the transformation in situ in a vitamin A metabolite that induces an anomaly hiperkeratinizarea sebochistilor differentiations with follicular canal). In this particular case is appealed, a cosmetic dermatology treatment and microsurgical suction of the cyst. 2) context hyperandrogenic hormonal abnormality. (2,3,6)
Acne Particular cases of acne treatment
Particular cases of acne treatment
Acne fulminans Although this rare form of ancnee may be encountered in practice in general dermatology with a high frequency of males between 13 and 18. Onset is abrupt at presternal, back and shoulders, with hemorrhagic ulcerative lesions, necrotic, fever, pol, and altered mental status ialtralgii General Biology finds proteinuria, circulating immune complexes, polinucleoza.Uneori progression to acne fulminans may be triggered by treatment isotretinoin, in which case corticosteroid therapy should be started at a dose necessary intrerupt.Este 0.5-1mg per kg body weight per zi.Unii author's association with NSAIDs or paracetamolului.Reintroducerea isotretinoin is gradual, with doses of 5-10 mg / kg of body is not possible reintroduction of isotretinoin zi.Daca can call on spironolactona.Foarte disulona or local measures are important in the vacuum treatment of cystic necrotic lesions, and especially psychological support. (2,3,6,)
Foliculitele with Gram negative They are the only cases that require sampling for culture and antibiogram according to which antibiotic to prescribe.
Piodermitele facial Occur especially in women with inflammatory abrupt onset, especially in the nose and obrajilor.Poate require general corticotherapy (0.5-1mg / kg), followed by NSAIDs, which are preferred by isotretinoin with higher anti-inflammatory effect ciclinelor.
Acne Particular cases of acne treatment Acne Conglobata Acne Conglobata represent 0.5-1% of cases of acne and is especially important for men with seborrhea. Because this type of acne cysts and tunnels which prevails out pus, and scarring are important, they must first be intent isotretinoin therapy (0.5-1mg / kg). Sometimes associated with corticosteroid therapy (0.5 mg / kg) and treatment of acne dermatochirurgical.Tratamentul in this form with your team to do plastician.Pacientul may need psychological support! (2,3,6)
Acne Escorial More common in women with psychiatric disorders or other neurotic tendency, this type of acne is well treated in collaboration with the psychiatrist. (Anxiolytics can be prescribed provided that the patient to accept collaboration with psychiatrist or psychologist).
Atrophic or hypertrophic scars of acne may benefit from peels to improve the appearance and performance chemicals pielii.Alegerea peels are done only by experienced dermatologists and plasticienii. (2,3,8)
Acne Particular cases of acne treatment Resistant or relapsing acne to isotretinoin It is generally caused by: 1) hereditary land hiperseboreic with parents acne (m2 mutations demonstrated the existence of an adenine-guanine instead of the cytochrome P450 gene in subjects acne, which leads to the transformation in situ in a vitamin A metabolite that induces an anomaly hiperkeratinizarea sebochistilor differentiations with follicular canal). In this particular case is appealed, a cosmetic dermatology treatment and microsurgical suction of the cyst. 2) context hyperandrogenic hormonal abnormality. (2,3,6)
Acne Particular cases of acne treatment
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