Saturday, January 8, 2011

Black papular dermatitis

Black papular dermatitis Black papular dermatitis is a common benign skin condition especially in the black population. It is characterized by multiple papules, small, hyperpigmented, asymptomatic girl. Histologically dermatitis Seborrheic dermatitis is similar. The condition has only cosmetic effects.
Generally requires no treatment. If deranzeaza dermatosis patient may try various methods such as microdermabrasion or surgical Cryosurgery. Premalignant condition and is not an excellent prognosis. Papular dermatitis lesions in black shows no tendency to spontaneous regression.
Pathogenesis and causes
Pathophysiology is not known black papulous dermatoses. Positive family history of disease suggests a genetic propensity. Dermatitis is probably closely genetically determined, 50% of patients had family history. It is believed to be caused by a defective development pilosebacee glands.
Signs and symptoms
Black papular dermatitis begins to develop in adolescence and is rare in people under 7 years. Disease incidence and number of lesions increased with age. Lesions usually starts after puberty. It is characterized by flat papules, numerous, strong, thin, brown-black that measures 1-5 mm. The lesions appear mainly on the face and forehead malaria, although it may occur on the neck, back and chest. There appears crustificarea, ulceration or scarring.
Diagnosis
Histological examination. Dermatitis lesions were black papulous histological appearance of seborrheic keratoses. Shows hyperkeratosis, irregular acanthosis, epidermal invagination filled with keratin and basal layer hyperpigmentation. Although most lesions are of acantotic and shows thick tracts between epidermal cells, they can present and mesh pattern. The differential diagnosis is made with the following conditions: melanocitici nevi, seborrheic keratosis, acrochordons, sebaceous adenoma.
Treatment
Do not need any treatment for this condition. Aggressive regimens were complicated by postoperative hyperpigmentation or hypopigmentation or scar formation. Keloid scar formation is also a possible complication, and therefore conservative treatment is recommended. Abrasive curettage with or without anesthesia, and electrodesicarea cryotherapy with liquid nitrogen followed by curettage have been shown effective. Laser therapy has also proved positive. Cream with lidocaine / prilocaine topical anesthesia is effective for. Evolution dermatoses is excellent because it is a premalignant condition and is not associated with other systemic disease. However, the lesions show no tendency to spontaneous healing and expand gradually.

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