Acneiform eruption

Acneiform eruption Acneiform eruptions may include cysts, comedones, papulopustule or nodules that resemble acne vulgaris. Occasionally it can lead to a wrong diagnosis. Acne related diseases occur due to a wide range of diseases, including infections, abnormal proliferation and drug reactions. These entities include nevus comedonicus, Pilar cysts eruptive, tuberous cleroza, steroid acne, cloracneea, acneiform drug eruptions, gram-negative folliculitis, eosinophilic pustular folliculitis, folliculitis with Pityrosporum, coccidomicoza, secondary syphilis, sporotrichoza, rosacea and perioral dermatitis.
Patients with similar injuries shows acneiform eruptions such as acne papulonodulii, pustules, and cysts comedoamele. Localizations include face, trunk and extremities. Evolution of lesions without evidence of etiology. These infections can form nodules, ulcers or crustificati. Systemic signs or symptoms may indicate a wrong diagnosis, some drug eruptions are associated with febrile disease and peripheral leukocytosis. It is important to note the herbal remedies used by patients, occupational exposure, pharmacological therapies administered.
Treatment varies depending on the disease involved and contains a wide range of methods, including excision, laser ablation, topical or oral antibiotics, topical retinoids or oral medication and trigger interruption.
Nevus comedonicus Represents an abnormal proliferation manifested as a group of aggregated open comedones. It consists of dilated eccrine or follicular openings with keratin plugs. It is also known as acneiform nevus side. It can be solitary, congenital or may appear later in life period as a result of occupational exposure. Disgnosticul differential includes family comedoamele comedomice dyskeratosis and linear formations associated with acne vulgaris or chronic exposure to sunlight. Rara these formations may raise suspicion of a neoplasm. Treatment is usually surgical excision or by laser ablation of carbon affected skin. Retinoids and topical therapy may give some benefits.
Pilar cysts eruptive It manifests as flesh-colored papules found on the face, chest, neck, groin, and armpits feze. It represents an anomaly velus follicles and can be hereditary. Histopathology showed a cyst containing skin epithelial keratinogen velus and material. These cysts may undergo spontaneous regression, or may degrade resulting in a foreign body granuloma. Treatment is often difficult. The practice of incision and drainage of lesions, but the risk of secondary scarring. Lactic acid and retinoids have proved useful in some cases.
Steroid acne It is seen as especially papulopustule monomorphic located on the trunk and extremities, without affecting the face. Typically occurs after administration of topical or systemic corticosteroids, including inhaled or intravenous therapy. The rash resolves upon discontinuation of therapy and may respond to acne medication.
Acne-cloracneea exposure to chemicals Exposure to halogenated aromatic compounds such as chlorinated and dibenzofurans diozina, inhalation, direct contact, ingestion of contaminated food or cause a rash with polymorphic and cysts called comedones cloracnee. Other features described include xerosis and skin pigmentation changes. Ophthalmic systems involving internal changes, nervous system and liver may also occur, some may be oncogenic cloracneeice. Treatment is difficult because cloracneea may persist for years, even without expansion. Chemicals that contain iodine, bromine and other halogens may induce acneiform eruptions similar to acne steroids. Induced form of iodine is extreme.
Acne medication Antibiotics can cause an acute generalized pustular eruption. Penicillins and macrolides are the most important in this class. Patients are often febrile, with leukocytosis and the rash does not involve comedones. Other antibiotics include co-tromoxazole involved, doxycycline, chloramphenicol. And other types of drugs can cause acneiform eruptions, including corticotropin, nystatin, isoniazid, itraconazole, hidroxicloroquina, naproxen, mercury, amineptina, lithium, chemotherapy.
Infectious cause acne Various infections can produce a model acneiform. Gram-negative folliculitis, a rash papulopustulara persistence may be a complication in patients with prolonged treatment for acne vulgaris or rosacea. It is more common in men. Cultures from these lesions showed gram-negative bacilli and cocci, including Escherichia coli, Klebsiella, Enterobacter, Proteus. Treatment consists of antibiotics with adequate coverage range. Isotretinoin may be an effective alternative.
Acne of the Pityrosporum folliculitis Pityrosporum folliculitis is an infection of the follicle caused by a fungus, Malassezia furfur, and formerly called Pityrosporum ovale, a skin commensal. His debut on the trunk and upper extremities in adolescence and young adulthood. Unlike acne vulgaris, is itching, does not include comedones and respond to antifungal therapy. The biopsy can be observed along the hyphae of the fungus keratinogen material. Treatment includes topical or systemic antifungal therapy.
Eosinophilic pustular folliculitis in acne It is a disease of unknown etiology that manifests as a pruritic eruption papulopustulara appellant on the face, trunk and extremities. Histopathology showed eosinophilic infiltrate composed of eosinophils perifolicular and pustules. It is described in immunosuppressed patients and children with HIV. Patients have peripheral eosinophilia and leukocytosis. Treatments vary widely. Options include topical and systemic corticosteroids, oral antibiotics, indomethacin, dapsaona, osotretinoin and PUVA.
Perioral dermatitis Perioral dermatitis is a disease of unknown etiology, seen especially in white women, young as papulopustule erythematous base. The eruption is localized predominantly perioral, avoiding the edge of the lip skin, but including perinazale and periorbital areas. Biopsies are rarely performed and show similar changes in rosacea. The etiology is unknown, and this triggers include Demodex, topical or inhaled corticosteroids, moisturizers, fluorinated compounds and contact dermatitis. Includes discontinuing steroid therapy and initiation of antibiotic therapy with metronidazole.