Congenital hip dysplasia Pathogenesis

Congenital hip dysplasia

    * Introduction
    * Pathogenesis
    * Signs and symptoms
    * Diagnosis
    * Treatment
Pathogenesis
Etiologic factors of hip dysplasia are not known, but the condition seems to be associated with a number of factors such as genetic, racial, sex and intrauterine position of the child, oligohidramonosul, cerebral palsy, myelomeningocele, artrogripoza. Racial differences are an etiological factor for hip dysplasia, so the condition is more common among natives of Lapland, American, black and Chinese populations. Compared to the genetic predisposition seems to exist a 10-fold for children with parents suffering from hip dysplasia to the disease. Other factors incriminated are female, first child. Over 80% of patients are female. Other musculoskeletal disorders of intrauterine or superposition of states malpozitiei as metatarsus adductus and torticollis are reported to be associated with hip dysplasia. Dysplasia is more common on the left side than on the right side due to its location on the child's mother left in front of the sacrum. Children from cultures which wraps your baby close, forcing adductie thighs. Development of hip dysplasia involves abnormal growth of it. Associated ligament laxity.
Anatomy of hip joint development: Develop normal balance dintrun single block of cartilage that separates the acetabular and femoral components at 7-8 weeks of gestation. Shape characteristic of balance is the result of contact between acetabulu and femur during growth. At birth the acetabulum is a small bone and a cartilage component of the sea. In the first 6 weeks postnatally succeptibil acetabulum is modeling. If you are in a position the femoral head in acetabulum abnormal balance is abnormal.
Normal growth depends on increasing the acetabulum of the normal epiphyseal cartilage of the triradiate and three centers of ossification of the acetabular portion of the pubis, iliac and ischiatic. Normal growth depends on increasing the interstitial apozitionala acetabulum from acetabulum normal. This spherical femoral head in acetabulum is critical to stimulate normal development of the acetabulum. Anatomy dislocated hip, especially after a few months include forming a bridge called neolimbus. Closed reduction is not successful in late presentations of multiple secondary barriers to the reduction. These include abductorului and psoas contraction, ligament tears, transverse acetabular ligament, and capsular constriction pulvinara. The long dislocations interpozitia labrum may interfere with the reduction.