Bursitis Pathogenesis and causes

Bursitis

    * Introduction
    * Pathogenesis and causes
    * Signs and symptoms
    * Diagnosis
    * Treatment

Pathogenesis and causes
Cause inflammation of the synovial cell proliferation exchanges and the formation of collagen and increases production of fluid. A more permeable capillary membrane protein allows fluid entry. Sealers scholarship can be replaced by granular tissue and then fibrous tissue. Bleeding occur. In septic arthritis, trauma usually cause local inoculation of bacteria in stock waging the inflammatory process. Bursitis can occur by many causes including acute trauma, chronic friction, storage of crystals in gout and pseudogout, systemic infection and disease. Bursal evolutionary stages: acute, chronic and recurrent. During the first phase of local inflammation occurs and is diminished synovial fliudul forcing painful joint movements. Bursitis Chronic pain leads to further weakening ligementelor and tendons and cause them to rupture.
Etiology bursitis. General bursitis. Inflammation can occur through numerous scholarship causes: acute trauma, chronic friction, storage of crystals in gout and pseudogout, systemic infection and disease: rerumatoida arthritis, ankylosing spondylitis, psoriatic arthritis, scleroderma, lupus erythematosus, pancreatitis, Wipple disease, oxaloza, uremia, osteoartropatia pulmonary hypertrophic, idiopathic hypereosinophilic syndrome. Septic bursitis. Occurs through direct introduction of microorganisms through traumatic injury or continuity from one cell 70% of cases. Less profound scholarship is the result of infection or septic arthritis, bacteraemia, 10% of cases. Bodies are encountered causing the Staphylococcus aureus-80% of cases, followed by streptococci. Other organisms include Mycobacterium tuberculosis or nonspecific-strains, Candida fungi, and algae, Prototheca wickerhamii. Predisposing factors include diabetes, alcoholism, steroid therapy, uremia, trauma and skin diseases. History of inflammation of the exchange noninfectioasa also increases the risk of septic bursitis.
Anatomy scholarships. There are two types of scholarships: constant and adventiale. Both types can be affected. Scholarships constants: -Appeared in embryological development Are lined by endothelium- -Are located between tendon and bone or skin Synovial luchid-containing collagen and proteoglycans rich. Scholarships adventiale: -Occurring in late stages of life TRUMA response to repeated or constant friction and pressure Not endothelium- -Not contain synovial fluid.