The anomaly is usually discovered during a routine fetal ultrasoundinvestigation or obstetric indication for investigation after finding anincreased level of alpha-fetoprotein.
Exteriorized organs are covered by a membrane-peritoneum.Unlike gastroschisis, peritoneum bodies appear to effect longexposure to amniotic fluid and then the external environment.However, if the hole is too small and organs are in large amountsthey can suffer damage from ischemia by compression ofmesenteric vessels. The consequences include prolongedischemic necrosis of organs, with their perforation and peritonitis.This condition is very dangerous for the child's life.
Complications include mechanical omphalocele volvulareaintestinal loops in the peritoneum with secondary ischemia. Alsoperitoneum may break through the bodies of its high compressionor inflammatory damage after bacterial infection. Bacterialinfection and sepsis cause peritonitis.
Other complications include impaired absorption starvationthrough the intestines. Infections caused over 13% of neonatal deaths. During pregnancy than fetuses with omphalocele half will die. At delivrenta omfalocelele undiagnosed can create problemsfor antenatal mother and baby by damage organs exposed. Inthese cases but also in the cesarean section is recommendedantenatal discovered.
Children with large omphalocele presents a central abdominaldefect with increased diameter. Omphalocele containing liver iscompletely. Chest and abdominal cavities are small andundeveloped. Restrictive lung disease and pulmonary hypoplasiaassociated with diminished chest cavity.
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