Hematoma rod

Hematoma rod Rod or epidural hematoma is a collection of blood located between the take-mater and cranial bones. Hematoma comes mostly from the meninges sized artery rupture or its branches. Less blood comes from the rupture or sinus meningeene parietal veins. Superhard hematoma is frequently associated with skull fractures and subdural hematomas develop faster than. These hematomas occur in about 3% of head trauma, subdural hematoma and compared it rarely accompany cortical lesions.
Topographic location of the hematoma rod Frequently, the hematoma is localized in the upper rod temporal convexity and have their origin in the bleeding artery and its branches meninges medium. The dura temporoparietala region is less adherence to the bone, which explains the frequency of hematoma rod at this level. Most patients have fractures scoamoase portion of the temporal bone, the damaged vessels path. Superhard hematoma are uncommon localized frontal or temporal occipitoparietala lower. Haematomas that develop in the posterior fossa are difficult to detect clinically, most are caused by surgical interventions at this level. The amount of blood that forms around the hematoma is 40-100 ml. In acute stage, a hematoma containing fresh blood meal with chiaguri loose and not adhere to the dura. In the subacute stage, chiagurile are firmer and adhere to the dura. Superhard chronic hematomas are rare, but when they exist are embedded in this phase. Hematoma rod separates from the inner surface dura of the skull. The elderly, firmly adhered to the dura and skull bones here to explain the relatively lower frequency of this disease in the elderly.
Clinical Manifestations - In the subacute stage Immediately after the traumatic event, may occur a temporary loss of consciousness, accompanied by agitation, hemiparesis, and pupillary changes. This is the time I of evolution. Then comes a period that stretches remissive a few hours to several days. During this period, the patient may be asymptomatic or present some minor symptoms or mild persistent headache, impaired balance, confusion, dizziness. II during the evolution is characterized by worsening neurological status. Altered state of consciousness through stages of confusion, anxiety, dizziness, stupor and finally coma. If not surgically intervene sooner, will be affected by deep coma with brain stem. Focal neurological signs are the neurological deficits like hemiparesis, hemiplegii, which are opposite the location of the hematoma. In advanced ophthalmic meet and phenomena as unilateral or bilateral mydriasis. Important vegetative disorders occur in deep coma. - In stage supra Clinical Evolution supra rod hematoma is very fast, inrervalul remissive no more. Sudden onset of Time II by coma and severe vegetative disorders. Bleeding is the rapid increase much in size and hematoma causing mass effect (compression of adjacent nerve formations). Around the hematoma, edematiaza nervous tissue. Transtentoriala employment phenomena and brainstem swinging, setting up an opposite side hemiplegia hematoma. - In the acute stage is seen between remission, but shorter (several hours). After this period, the patient suddenly and get worse in the state of coma. - Rarely meets the chronic stage, because the hematoma develops rapidly after the occurrence of causative trauma. One possibility of developing a hematoma to develop chronic lower bleeding vessel is injured nervous tissue that gives time to adjust to the compressive effect of the hematoma. Remissive range is 7-21 days and is getting worse very slowly.
Evolution and prognosis of hematomas rod The prognosis of these hematomas is better than other expansive intracranial lesions of traumatic origin. Evolution depends on the patient's condition at admission, his age, the earliness of diagnosis of underlying disease. Subacute and chronic forms have a poor prognosis and a better evolution than acute forms and supra.