Intracranial hemorrhages in hypertensive encephalopathy

Intracranial hemorrhages in hypertensive encephalopathy
Hypertensive encephalopathy occurs as a result of malignant hypertension, but can also be a complication of pregnancy, renal failure or pheochromocytoma. Men are more affected than women.

 
The most common clinical manifestations include occipital headache, retinopathy with papilloedema (including transient blindness), urermie, nausea, vomiting, confusion, vertigo, tinnitus, down to stupor and coma. These signs were attributed to cerebral edema and cerebral vessel spasm.

 
Other critical aspects of malignant hypertension and cardiac decompensation rapid alteration of renal function, and in some cases the reason for the presentation of the patient to the doctor can only oliguria.

 
Focal neurological signs are rare and, if present, suggests that heart attacks, bleeding, or transient ischemic attacks point to other forms of vascular disease.

 
Vascular lesion characteristic of malignant hypertension is fibrinoid necrosis of the small arteries and arterioles. Under proper antihypertensive treatment process can be stopped. At necropsy these patients were even found multiple clots in cerebral vessels. It seems that there are two independent processes that contribute to the signs and symptoms associated with: cerebral artery dilation and generalized arteriolar fibrinoid necrosis. Since the retina is the only tissue in the arteries and arterioles can be examined directly, repeatedly ophtalmoscope exam provides the opportunity to observe the progression of vascular effects of hypertension.

 
Malignant hypertension is a medical emergency. The main objectives of treatment are: - How many doctors to correct complications - Reduction of diastolic pressure by one third, but not less than 95 mmHg