Bone infarction - Signs and symptoms

Clinic heart disease in bone depends on the stage and location.Patients with medullary infarcts are usually asymptomatic. If present, symptoms are nonspecific in early stages of the disease.Patients with juxta-articular lesions shows instability. Localized pain is the most common symptom of heart attack and the juxta-articular bone marrow. These shows joint pain and limiting movement. Untreated affected bone will disintegrate with increasing pain and disability.In sickle cell patient may experience severe bone pain and abdominal pain associated with sickle cell crisis. In Perthes disease Legg-Calve-fracture and femoral head and groin pain is characteristic lameness and knee and thigh pain occurs. In spontaneous osteonecrosis of the tibia, pain in the joints genunhiului is located above the medial epicondyle. Osteonecrosis is commonly associated with spontaneous pain, tenderness, swelling and restriction of movement. In osteochondritis dissecans or missing symptoms vary, affecting the medial femoral condyle usually surface.
Traumatic Osteonecrosis:Osteonecrosis of the femoral head: the patients do not experience pain during the ischemic episode. Occult avascular osteonecrosis may be present 5 years before symptom onset. Patients may be asymptomatic or develop gradually and insidiously pain. May reduce movements and went limp. Pain can be cruel and sudden onset.Traumatic Osteonecrosis of the talus bone adjacent to cause osteoporosis. Osteonecrosis of the humeral head is usually a complication of fracture-dislocation or fracture anatomically end. In 10-15% of scaphoid bone fractures are complicated by osteonecrosis of the proximal pole of the scaphoid. Osteonecrosis of the capitate bone occurs after accidental or occupational trauma. Proximal portion of the bone is infarcted. After trauma or prolonged stress affects the heart and monthly bone, other carpal bones, navicular bone, cn = ondilul mandible, patella, scapula, and occasionally the glenoid region metatarsals. Osteonecrosis of the vertebral bodies-Kummell disease occurs several years after acute trauma. Determine the vertebral collapse around the middle.Generally lombarele are affected.
Spontaneous Osteonecrosis:This condition is Ahlback disease affect the knees, a distinct clinical entity that interest women more than men. It affects the medial femoral condyle and less often on the tibia. Untreated lead to joint collapse and sclerosis. Spontaneous osteonecrosis may occur navicularului syndrome Tarsal-Muller-Wiess, especially in women. The disease can be bilateral or asymmetric and associated with pathological fractures. It is associated with progression and severe pain and disability.
Osteonecrosis secondary Cushing's disease:Appears as a result of excessive levels of endogenous steroids.The high level of steroids is observed mostly in the vertebrae.Features are osteoporosis, osteoscleroza, collapse and fragmentation of bone associated with relatively normal joint space.Osteonecrosis in Gaucher disease:The disease affects the epiphysis and diaphysis is a complication of Gaucher disease and frequently associated with bone pain. In the long bones and sclerosis radiolucenta alternate bands. The bone will undergo sclerosis.Osteonecrosis of haemophilia:Osteonecrosis may complicate hemophilia and is described on the femoral head and talus. Myocardial intraosseous bone result from hemorrhage secondary to bone collapse or intracapsular hemorrhage and intra-articular pressure increase vascular compromise and osteonecrosis.Osteonecrosis of pancreatitis:Osteonecrosis is a well-known complication of pancreatitis, the chronic or inactive form. Describe the effect on distal femur and proximal tibia.Osteonecrosis in SLE:The pathogenesis of osteonecrosis in SLE is uncertain, and the role of steroids is speculative. Areas most affected are the femoral head, femoral CONDITIONS, tibial plateau and talus.Radiation Osteonecrosis:Internal and external exposure, accidental or diagnostic, therapeutic irradiation produces different bone changes, including growth interruption, infarction of bone, scoliosis and benign and malignant neoplasms. Bone changes are mediated by age and dose. Various parts of the skeleton respond differently to radiotherapy. The affected bones are the mandible, skull, sternum, shoulder. Radiation osteitis is manifested by osteoporosis, osteonecrosis of the jaw occurs in 1 year after irradiation and is the most common bone affected area due to its compact and low intake of blood. Shoulder necrosis occurs 10 years after irradiation. Sternum necrosis occurs after irradiation for breast cancer.
Complications of osteonecrosis:Abnormalities of cartilage, erosions and joint space collapse affects the joints. Cystic degeneration of bone infarction in areas usually in hollow shaft of bone. Sarcoma, malignant degeneration is a well-known complication of bone infarct from any cause. Men are more affected than women, patients ages 40-70 years.Typically the distal femur or proximal tibia are affected.